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Estradiol began at 22 pg/mL at Week 1, climbed to 48 pg/mL at Week 5, and reached 62 pg/mL by Week 9. This 182% increase tracks closely with the rise in total testosterone (485 → 1840 → 2240 ng/dL), consistent with peripheral aromatization of excess androgens into estrogens. No aromatase inhibitor is listed in the protocol, so there is no pharmacological brake on this conversion. Masteron Propionate has mild anti-estrogenic properties via aromatase competition, but clearly insufficient to prevent significant estradiol accumulation at these testosterone doses. The subjective libido data shows a dip at Weeks 7–8 (7.4 and 7.6/10 vs. a peak of 8.6 at Week 6), which temporally coincides with the period of highest estradiol accumulation, though this correlation is suggestive rather than conclusive. The monotonic rise across three timepoints with a clear mechanistic explanation supports high confidence.